Serum Asymmetric Dimethylarginine (ADMA)

Chronic Angiotensin II Receptor Blockade Reduces (Intra)Renal Vascular Resistance in Patients with Type 2 Diabetes

Oxidative Stress / Humans / Kidney / Blood Pressure / Renin Angiotensin Aldosterone System / Nitric oxide / Female / Male / Clinical Sciences / Aged / Middle Aged / Imidazoles / Type 2 Diabetes Mellitus / Angiotensin II / Serum Asymmetric Dimethylarginine (ADMA) / Prostaglandin E2 / The American / Treatment Effect / Vascular Resistance / Nitric oxide / Female / Male / Clinical Sciences / Aged / Middle Aged / Imidazoles / Type 2 Diabetes Mellitus / Angiotensin II / Serum Asymmetric Dimethylarginine (ADMA) / Prostaglandin E2 / The American / Treatment Effect / Vascular Resistance

α-Lipoic acid activates dimethylarginine dimethylaminohydrolase in cultured endothelial cells

Cardiovascular disease / Gene expression / Endothelial Cells / Mice / Animals / Risk factors / Enzyme / Endothelial Function / Endothelial cell / Arginine / Risk Factors / Culture Media / Serum Asymmetric Dimethylarginine (ADMA) / Binding Site / Biochemistry and cell biology / Enzyme Induction / Risk factors / Enzyme / Endothelial Function / Endothelial cell / Arginine / Risk Factors / Culture Media / Serum Asymmetric Dimethylarginine (ADMA) / Binding Site / Biochemistry and cell biology / Enzyme Induction

Chronic Angiotensin II Receptor Blockade Reduces (Intra)Renal Vascular Resistance in Patients with Type 2 Diabetes

Oxidative Stress / Type 2 Diabetes / Humans / Kidney / Blood Pressure / Renin Angiotensin Aldosterone System / Nitric oxide / Female / Male / Clinical Sciences / Aged / Middle Aged / Imidazoles / Type 2 Diabetes Mellitus / Angiotensin II / Serum Asymmetric Dimethylarginine (ADMA) / Prostaglandin E2 / The American / Treatment Effect / Vascular Resistance / Renin Angiotensin Aldosterone System / Nitric oxide / Female / Male / Clinical Sciences / Aged / Middle Aged / Imidazoles / Type 2 Diabetes Mellitus / Angiotensin II / Serum Asymmetric Dimethylarginine (ADMA) / Prostaglandin E2 / The American / Treatment Effect / Vascular Resistance

Dimethylarginine dimethylaminohydrolase-2 deficiency promotes vascular regeneration and attenuates pathological angiogenesis

Angiogenesis / Serum Asymmetric Dimethylarginine (ADMA)

Dimethylarginine Dimethylaminohydrolase Regulates Nitric Oxide Synthesis: Genetic and Physiological Evidence

Genetics / Physiology / Metabolism / Cardiovascular / Control / Regulation / Cardiovascular Physiology / Transgenic Mice / In Vitro / Humans / Hemodynamics / Mice / Blood Pressure / Nitric oxide / Resistance / Animals / Cell / Plasma / Transgenic Animals / Volume / ATP synthase / Expression / Gene / Reduction / Risk factors / Vascular / Support / Enzyme / Clinical Sciences / Cultured meat / In Vivo / Activity / Nitric Oxide Synthase / Endothelial cell / Public health systems and services research / Endogenous / Circulation / Arginine / Risk Factors / Transfection / Transgenic / ENDOTHELIUM / Serum Asymmetric Dimethylarginine (ADMA) / Vasodilation / Level / Urinary / Gene Transfer / Systolic blood pressure / Vascular Resistance / Mm / Contractility / Regulation / Cardiovascular Physiology / Transgenic Mice / In Vitro / Humans / Hemodynamics / Mice / Blood Pressure / Nitric oxide / Resistance / Animals / Cell / Plasma / Transgenic Animals / Volume / ATP synthase / Expression / Gene / Reduction / Risk factors / Vascular / Support / Enzyme / Clinical Sciences / Cultured meat / In Vivo / Activity / Nitric Oxide Synthase / Endothelial cell / Public health systems and services research / Endogenous / Circulation / Arginine / Risk Factors / Transfection / Transgenic / ENDOTHELIUM / Serum Asymmetric Dimethylarginine (ADMA) / Vasodilation / Level / Urinary / Gene Transfer / Systolic blood pressure / Vascular Resistance / Mm / Contractility

Dimethylarginine Dimethylaminohydrolase Regulates Nitric Oxide Synthesis Genetic and Physiological Evidence

Genetics / Physiology / Metabolism / Cardiovascular / Control / Regulation / Cardiovascular Physiology / Transgenic Mice / In Vitro / Humans / Hemodynamics / Mice / Blood Pressure / Nitric oxide / Resistance / Animals / Cell / Plasma / Transgenic Animals / Volume / ATP synthase / Expression / Gene / Reduction / Risk factors / Vascular / Support / Enzyme / Clinical Sciences / Cultured meat / In Vivo / Activity / Nitric Oxide Synthase / Endothelial cell / Public health systems and services research / Endogenous / Circulation / Arginine / Risk Factors / Transfection / Transgenic / ENDOTHELIUM / Serum Asymmetric Dimethylarginine (ADMA) / Vasodilation / Level / Urinary / Gene Transfer / Systolic blood pressure / Vascular Resistance / Mm / Contractility / Regulation / Cardiovascular Physiology / Transgenic Mice / In Vitro / Humans / Hemodynamics / Mice / Blood Pressure / Nitric oxide / Resistance / Animals / Cell / Plasma / Transgenic Animals / Volume / ATP synthase / Expression / Gene / Reduction / Risk factors / Vascular / Support / Enzyme / Clinical Sciences / Cultured meat / In Vivo / Activity / Nitric Oxide Synthase / Endothelial cell / Public health systems and services research / Endogenous / Circulation / Arginine / Risk Factors / Transfection / Transgenic / ENDOTHELIUM / Serum Asymmetric Dimethylarginine (ADMA) / Vasodilation / Level / Urinary / Gene Transfer / Systolic blood pressure / Vascular Resistance / Mm / Contractility

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